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Frontiers | TAK1-TABs Complex: A Central Signalosome in Inflammatory  Responses | Immunology
Frontiers | TAK1-TABs Complex: A Central Signalosome in Inflammatory Responses | Immunology

Th17 Cells: Biology, Pathogenesis of Autoimmune and Inflammatory Diseases,  and Therapeutic Strategies - ScienceDirect
Th17 Cells: Biology, Pathogenesis of Autoimmune and Inflammatory Diseases, and Therapeutic Strategies - ScienceDirect

Nuclear factor kappa B (NF-κB) in multiple sclerosis pathology: Trends in  Molecular Medicine
Nuclear factor kappa B (NF-κB) in multiple sclerosis pathology: Trends in Molecular Medicine

Frontiers | The IL-23/IL-17 Pathway in Inflammatory Skin Diseases: From  Bench to Bedside | Immunology
Frontiers | The IL-23/IL-17 Pathway in Inflammatory Skin Diseases: From Bench to Bedside | Immunology

Nuclear factor kappa B (NF-κB) in multiple sclerosis pathology: Trends in  Molecular Medicine
Nuclear factor kappa B (NF-κB) in multiple sclerosis pathology: Trends in Molecular Medicine

Interferon regulatory factor 5 and nuclear factor kappa‐B exhibit  cooperating but also divergent roles in the regulation of pro‐inflammatory  cytokines important for the development of TH1 and TH17 responses - Stein -
Interferon regulatory factor 5 and nuclear factor kappa‐B exhibit cooperating but also divergent roles in the regulation of pro‐inflammatory cytokines important for the development of TH1 and TH17 responses - Stein -

PDF) NF-κB signaling in inflammation
PDF) NF-κB signaling in inflammation

Enhanced NF-κB signaling in type-2 dendritic cells at baseline predicts  non-response to adalimumab in psoriasis | medRxiv
Enhanced NF-κB signaling in type-2 dendritic cells at baseline predicts non-response to adalimumab in psoriasis | medRxiv

The Cytokine IL-17A Limits Th17 Pathogenicity via a Negative Feedback Loop  Driven by Autocrine Induction of IL-24 - ScienceDirect
The Cytokine IL-17A Limits Th17 Pathogenicity via a Negative Feedback Loop Driven by Autocrine Induction of IL-24 - ScienceDirect

Th17/IL-17 induces endothelial cell senescence via activation of NF-κB/p53/Rb  signaling pathway | Laboratory Investigation
Th17/IL-17 induces endothelial cell senescence via activation of NF-κB/p53/Rb signaling pathway | Laboratory Investigation

IJMS | Free Full-Text | Th17 Cells and the IL-23/IL-17 Axis in the  Pathogenesis of Periodontitis and Immune-Mediated Inflammatory Diseases |  HTML
IJMS | Free Full-Text | Th17 Cells and the IL-23/IL-17 Axis in the Pathogenesis of Periodontitis and Immune-Mediated Inflammatory Diseases | HTML

Targeting NF-κB pathway for the therapy of diseases: mechanism and clinical  study | Signal Transduction and Targeted Therapy
Targeting NF-κB pathway for the therapy of diseases: mechanism and clinical study | Signal Transduction and Targeted Therapy

Frontiers | Nuclear Factor κB (NF-κB)–Mediated Inflammation in Multiple  Sclerosis | Immunology
Frontiers | Nuclear Factor κB (NF-κB)–Mediated Inflammation in Multiple Sclerosis | Immunology

NF-κB control of T cell development | Nature Immunology
NF-κB control of T cell development | Nature Immunology

NF-κB signaling in inflammation | Signal Transduction and Targeted Therapy
NF-κB signaling in inflammation | Signal Transduction and Targeted Therapy

TRIM25 Promotes TNF-α–Induced NF-κB Activation through Potentiating the  K63-Linked Ubiquitination of TRAF2 | The Journal of Immunology
TRIM25 Promotes TNF-α–Induced NF-κB Activation through Potentiating the K63-Linked Ubiquitination of TRAF2 | The Journal of Immunology

Factors regulating Th17 cells: a review
Factors regulating Th17 cells: a review

A Case for Targeting Th17 Cells and IL-17A in SARS-CoV-2 Infections | The  Journal of Immunology
A Case for Targeting Th17 Cells and IL-17A in SARS-CoV-2 Infections | The Journal of Immunology

Frontiers | Th17 and Cognitive Impairment: Possible Mechanisms of Action |  Frontiers in Neuroanatomy
Frontiers | Th17 and Cognitive Impairment: Possible Mechanisms of Action | Frontiers in Neuroanatomy

Enhanced NF-κB signaling in type-2 dendritic cells at baseline predicts  non-response to adalimumab in psoriasis | medRxiv
Enhanced NF-κB signaling in type-2 dendritic cells at baseline predicts non-response to adalimumab in psoriasis | medRxiv

NF‐κB and HIF crosstalk in immune responses - D'Ignazio - 2016 - The FEBS  Journal - Wiley Online Library
NF‐κB and HIF crosstalk in immune responses - D'Ignazio - 2016 - The FEBS Journal - Wiley Online Library

The non-canonical NF-κB pathway in immunity and inflammation | Nature  Reviews Immunology
The non-canonical NF-κB pathway in immunity and inflammation | Nature Reviews Immunology

IL-17 and Th17 Cells in Atherosclerosis | Arteriosclerosis, Thrombosis, and  Vascular Biology
IL-17 and Th17 Cells in Atherosclerosis | Arteriosclerosis, Thrombosis, and Vascular Biology

Frontiers | Cell Type-Specific Roles of NF-κB Linking Inflammation and  Thrombosis | Immunology
Frontiers | Cell Type-Specific Roles of NF-κB Linking Inflammation and Thrombosis | Immunology

Cytokine production by Th17 cells in CF Pathway Map - PrimePCR | Life  Science | Bio-Rad
Cytokine production by Th17 cells in CF Pathway Map - PrimePCR | Life Science | Bio-Rad

The roles of the classical and alternative nuclear factor-kappaB pathways:  potential implications for autoimmunity and rheumatoid arthritis |  Arthritis Research & Therapy | Full Text
The roles of the classical and alternative nuclear factor-kappaB pathways: potential implications for autoimmunity and rheumatoid arthritis | Arthritis Research & Therapy | Full Text

Frontiers | Th17 CD4+ T-Cell as a Preferential Target for HIV Reservoirs |  Immunology
Frontiers | Th17 CD4+ T-Cell as a Preferential Target for HIV Reservoirs | Immunology

IL-23 and IL-1β Drive Human Th17 Cell Differentiation and Metabolic  Reprogramming in Absence of CD28 Costimulation: Cell Reports
IL-23 and IL-1β Drive Human Th17 Cell Differentiation and Metabolic Reprogramming in Absence of CD28 Costimulation: Cell Reports